Yet, most Americans get approximately 8% of their calories from seed oils. 7 When I interviewed Tucker Goodrich, who developed an IT risk management system used by two of the largest hedge funds in the world, then transitioned into medical research, he explained that animals typically develop cancer once the linoleic acid in their diet reaches 4% to 10% of their energy intake. There’s virtually nothing more destructive to your body than seed oils in producing heart disease, cancer, age-related macular degeneration, diabetes, obesity and dementia. Linoleic Acid Drives Chronic Diseases, Worsens COVID-19 Paul Saladino, a physician journalist, also explained in a podcast that linoleic acid “breaks the sensitivity for insulin at the level of your fat cells,” 6 essentially making them more insulin sensitive - and, since your fat cells control the insulin sensitivity of the rest of your body by releasing free fatty acids, you end up with insulin resistance. OXLAMs are also toxic to the liver and are associated with inflammation, fibrosis and fatty liver disease in humans. Because there are so many reactive oxygen species it leads to the development of insulin resistance at the cellular level. Their atherosclerosis and thrombogenic actions are especially concerning because they can produce strokes and clots, however metabolic dysfunction can also occur.ĭuring the lipid peroxidation cascade caused by the excess consumption of omega-6 seed oils, PUFAs accumulate in your cell membranes, leading to a peroxidation reaction. OXLAMs (oxidized linoleic acid metabolites) create a perfect storm, as they are cytotoxic, genotoxic, mutagenic, carcinogenic, atherogenic and thrombogenic, according to Knobbe. Chris Knobbe, an ophthalmologist and the founder and president of the Cure AMD Foundation. “Most of this linoleic acid, when it oxidizes, it develops lipid hydroperoxides and then these rapidly degenerate into … oxidized linoleic acid metabolites,” says Dr. Not only are most of the omega-6s you eat, including seed oils, damaged and oxidized through processing, but even if they are unheated and pristine when consumed in any but small amounts, your body degrades them into free radicals that damage virtually every tissue in your body. These fats become embedded in your cell membranes and stay there for about seven years, wreaking havoc on your health. “The biological functions of most of these genes and their mammalian orthologs have not been fully elucidated,” the study says - basically, we still don’t understand why these genes exist.But most health “experts” simply do not understand that seed oils are even worse than sugar. Those genes also “not only prevents excessive fat accumulation in primary and ectopic fat depots but also improve the health and extends the lifespan” of the worms. In the worms that were given the high-fructose diet, the three genes that “suppress” obesity also “partially suppressed the detrimental effects” of that diet. Researchers gave some of the worms a normal diet, and others an unhealthy, high-fructose diet to mimic “diet-induced obesity,” and screened the worms for those genes, using what the study described as a “high-throughput genetic screening strategy.”Īfter comparing their findings with other genome-wide association studies that have been performed on mice and on humans, the researchers were able to identify 14 genes that cause obesity, and three that prevent it. Then they screened them through worms that share most of their genes - more than 70 per cent - with humans. causation, the researchers identified 340 genes that they knew from previous studies were more likely to show up in obese people, and built a “library” of those genes. In an attempt to overcome the barrier of correlation vs.
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